Gout
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Gout is a form of arthritis that affects mostly middle-aged men and
postmenopausal women. After 3 weeks of adding Wobenzym®
to conventional gout therapy, they saw an improvement of
94.1% compared to only 47.3% with only conventional therapy. So when you look at the research, you could say that Wobenzym is a good therapy for all forms of arthritis including osteoarthritis, rheumatoid arthritis, as well as psoriatic arthritis, juvenile chronic arthritis and even gouty arthritis. From FREQUENTLY ASKED QUESTIONS, with answers by Joseph J Collins, RN, ND Read the special section: JOINT PAIN & WOBENZYM® |
What the literature says about Systemic Enzyme Support and:
Systemic enzyme therapy in the gout treatment
Kovalenko V.N., Siniatchenko O.V., Ignatchenko G.A., Terzov A.I.,
Grin V.K., Lauschkina E.M.Systemic enzyme therapy in the gout
treatment. Ukrainskii kardiologitschnyi zurnal 1998: 1, 53-56.
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male patients suffering from primary gout were observed. Patients
received non-steroidal antiinflammatory drugs (diclofenac sodium,
movalis, phelden, indomethacin) and allopurinol (milurit).
19 patients, aged
28-69 years (mean 48.9)
17 patients, aged
29-71 (mean 50.2)
Patients in the second group received Wobenzym (5 dragees 3 times a
day for 1 week, then 4x3 dragees for 7 days, and finally 3x3 dragees
for 1 month) in the complex therapy.
Before therapy and after 3 weeks, purine parameters were determined
in the blood – uric acid, oxypurinol, xanthine oxidase,
5-nucleotidase, adenosine deaminase, glycine, glutamine, and
aspartic acid.
The
efficacy of articular syndrome treatment in patients receiving
Wobenzym reached 94.1% in comparison to 47.3% in the control group.
Even more obvious were differences regarding renal syndrome.
While the conventional gout treatment led to the decrease of only
uric acid and xanthine oxidase in the blood, inclusion of Wobenzym
into the therapy caused decrease of oxypurinol, 5-nucleotidase and
adenosine deaminase level. Of special interest are data concerning
the investigation of serum dynamic interphase tensometry before and
after treatment of patients in both groups. Administration of
allopurinol and non-steroidal antiinflammatory drugs evoked a
suppression of surface tension in the region of short, middle, and
long surface life-time. This might be caused by the accumulation of
substances with surfactant properties.