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Lymphedema

Lymph is the fluid that is formed from the fluids that bathe and surround the cells in the tissues of the body. This fluid is carried back to the circulation through lymphatic vessels. When these vessels do not drain properly the fluid collects in the tissues causing edema, or more specifically lymphedema.
So, lymphedema is the swelling of tissue due to retention of fluid in the lymph vessels. Lymphedema can have many causes affects 175 million people worldwide, and can lead to infection, pain, severe disability and even increased risk of cancer. It is a disorder which is traditionally very difficult to treat. So you can realize why I was pleased to read the studies that were done with Wobenzym® treating lymphedema.
What I found, was that a number of studies specifically looked at the affect that Wobenzym® has on lymphedema. It was noted that there was significant reduction in pain and swelling with a lower risk of infections. Even primary lymphedema (hereditary lymphedema) showed significant improvement.
                               From FREQUENTLY ASKED QUESTIONS, with answers by Joseph J Collins, RN, ND
                               Read the special section: :
CARVIOVASCULAR AND LYMPHATIC SYSTEMS & WOBENZYM®

What the literature says about Systemic Enzyme Support and:

Lymphedema
Diagnosis and treatment of Lymphedema

Wald M. Diagnosis and treatment of lymphedema. Interní medicína pro praxi 2003, č. 8, str. 415-417.  

Definition and etiology of lymphedema is briefly explained as well as stages of the disease and possible complications. Clinical finding and suitable examination methods are described. Determination of lymphedema diagnosis is schematically depicted. Medicamentous treatment using systemic enzyme therapy preparation Wobenzym is mentioned in addition to physiotherapy. Poster Reference Number 39.  

Lymphedema.  

Kafková H., Kojanová M. Lymphedema. Postgraduální medicína 2003, 5 (6), pp. 626 – 633 (Department of Dermatovenerology, 1st Medical School of Charles University, Prague)  

The authors describe in detail lymphedema as an edema owing to an impaired lymph transport caused by interruption of lymphatic routes, infection or congenital abnormity. Moreover, possible complications of lymphedema, mainly development of secondary infections, are mentioned. An early and correct diagnosis using lymphoscintigraphy, ultrasound and CT examination is stressed both in primary and secondary lymphedema. Systemic enzyme therapy using combined mixtures of proteolytic enzymes (chymotrypsin, trypsin, bromelain, papain etc.) represents an effective treatment modality in pharmacotherapy of lymphedema. Enzyme therapy can improve already indurated and sclerotized tissue even in fully developed lymphedema. Based on the results of clinical trial in patients after breast cancer surgery it has been suggested that beginning of enzyme treatment immediately after surgery is an optimum therapeutic approach.
Poster Reference Number 40.  

Systemic enzyme therapy and complex decongestive therapy in the patients with primary and secondary lymphedema of lower extremity  

Dzupina A., Morvay P., Dzupinova M. Systemic enzyme therapy and complex decongestive therapy in the patients with primary and secondary lymphedema of lower extremity. Lymfo 2000, Praha 13. - 14. 10. 2000. Praktická flebologie - supplement 2000, Roè. IX, str. 23-27. (17-13-2)-(17-12-1)  

Summary:
The goal of this trial is the evaluation of Wobenzym efficiency as an additional therapy to the standard treatment of lymphedema. Two groups of patients were observed: 12 women with primary lymphedema of lower extremity (group I.) and 20 women suffering by secondary lymphedema following erysipelas. The diagnosis of lymphedema was confirmed by clinical examination, duplex sonography, radionuclide lymphoscintigraphy, and by CT and MRI eventually. Complex decongestive therapy (CDT) was used as a standard treatment including manual lymph drainage, sequential gradient pneumatic therapy, a bandaging and special exercises with aquatherapy. After 4 weeks of CDT both groups were randomly divided in two halves. The subgroups I. A, II. A continue the treatment by CDT only. Subgroups I. B and II. B received Wobenzym 3x3 tablets daily additionally to CDT. The treatment of all groups continued following 6 weeks.
During the observation subjective symptoms were estimated by questionnaire method, extremity volume differences and centripetal fraction of venous flow were measured. Serum levels of liver enzymes, creatinine and minerals were checked.
In the groups receiving Wobenzym a statistically significant amelioration of all objectives were evident comparing to groups treated by CDT only. The comparison of control visit results after 4 and 10 weeks of treatment - subjective symptoms I. B p < 0,0004, II. B p < 0,0002, extremity volumes I. B p < 0,005, II. B p < 0,003, centripetal fraction of venous flow I. B p < 0,005, II. B p < 0,003.
The treatment results in the patients with secondary lymphedema were better then the results in the patients with primary lymphedema. Nevertheless the results of the primary lymphedema patients showed statistical significance what is remarkable in this type of lymphedema with a generally known worse curability.
Key words: lymphedema - complex decongestive therapy - Wobenzym
Poster Reference Number 41.  

Orally administered proteases in aesthetic surgery.  

Dusková M, Wald M. Orally administered proteases in aesthetic surgery. Aesthetic Plast Surg. 1999 Jan-Feb;23(1):41-4.  

Increasing demand for shortening the sequel period after aesthetic surgery hasled to comparative testing of optional approaches. Systemic enzyme therapy withits pharmacological effects represents a preventive and curative option forinflammatory process including healing. Excellent results were presented, namely,in the treatment of secondary lymphoedema. The incidence of hematoma, edema, and pain was followed, and the results were compared in a randomized group of 20patients with upper eyelid blepharoplasty treated with proteases (Wobenzym drg)and in a similar group treated with systemic antiedema and hemostyptic therapy(Dicynone drg and Reparil drg). No undesirable side effects were observed. Inaddition, proteases apparently have no limitation for patients with the risk ofconcurrent cardiovascular, hepatic, or renal diseases.
External Link:
PMID: 10022937
Poster Reference Number 42.  

Proteolytic enzymes in lymphedema therapy  

Dzupina A., Morvay P., Dzupina M. Proteolytic enzymes in lymphedema therapy. 41st Annual World Congress - ICA'99, International College of Angiology, Sapporo, Japan, July 3-10, 1999, Scientific Posters pp. 76 601 KA  Department of Internal Medicine,City Hospital, Bardejov, Slovak Republic Department of Immunoallergology, City Hospital, Bardejov, Slovak Republic   

Purpose: The goal of this study was to establish the effect of Wobenzym in lymphedema therapy.
Material and Methods: Clinical examination, duplex sonography, radionuclide lymphoscintigraphy, CT and MRI  established the diagnosis of lymphedema in 50 patients (12 primary, 38 secondary). After 4 weeks of standard treatment consisting of manual lymph drainage, bandaging, sequential gradient pneumatic therapy (Pneuven Bevuk), and special exercise therapy including aqua therapy, 25 randomly selected patients continued in standard therapy and 25 received 9 Wobenzym tablets daily for 6 weeks. A Wobenzym tablet contains pancreatin 100 mg, bromelain 45 mg, papain 60 mg, triacylglycerol lipase 10 mg, amylase 10 mg, trypsin 24 mg, chymotrypsin 1 mg and rutoside 50 mg. Patients had monitoring for common symptoms of pain, feelings of heaviness and decreased mobility; extremity volume changes; and, serum levels of liver enzymes, creatinine and minerals. Duplex sonography was used to determine the centripetal frac­tion of venous flow.
Results: Wobenzym-treated patients noted significant beneficial effects in subjective symptoms (56%, p < 0.005), re­duction of extremity volume (55%, p <0.005), duplex sonography measurements (43%, p < 0.005) compared with the non-Wobenzym group.
Conclusion: Adding Wobenzym to complete standard therapy significantly improves subjective and objective parame­ters of lymphedema. No adverse effects or significant changes in laboratory data were observed.
Poster Reference Number 43.  

Wobenzyme and diuretic therapy in lymphedema after breast operation.  

Korpan MI, Fialka V. Wobenzyme and diuretic therapy in lymphedema after breast operation. Wien Med Wochenschr. 1996;146(4):67-72; discussion 74.  

The authors of this clinical study report the results of a controlled clinicaltrial in randomized parallel groups (Wobenzym vs. diuretics) of 55 femalepatients suffering from brachial arm lymph edema subsequent to ablatio mammae.All patients received manual and machine lymph drainage as well as gymnastics as concomitant therapy. After 7 weeks of therapy the results of the volometricassessments of the arm, the circumference of the arm and the skinfold thicknessshowed significant improvements compared to diuretics. In addition, the patients receiving Wobenzym reported a significantly higher proportion of patients free ofpain compared to the diuretics patients. Overall safety assessment results aresatisfactory thus resulting in a superior benefit/risk relation of the Wobenzymgroup.
External Link:
PMID: 8650941
Poster Reference Number 44.  

Efficacy and tolerability of proteolytic enzymes as an anti-inflammatory agent in lymphoedema after axillary dissection due to mammary cancer  

Kasseroller R., Wenning H.G. Efficacy and tolerability of proteolytic enzymes as an anti-inflammatory agent in lymphoedema after axillary dissection due to mammary cancer. The European Journal of Lymphology, 2002-2003, Vol. 10, No. 37-38, pp. 18-26,  

Lymphoedema is a chronic disease caused by the damage of lymphatic vessels due to surgical treatment and/or radiotherapy (secondary). Another cause is the malformation or lack of lymphatic vesels (primary).
The aim of the study was to demonstrate the efficacy and tolerability of the proteolytic enzyme combination preparation Wobenzym in additional reduction of arm volume (primary criterion) in patients with secondary lymphedema after dissection of axillary nodes due to mammary cancer. Secondary criteria were improvement of the skinfold thickness, CRP values, tension, and global judgement of the efficacy by both investigator and patient.
The study population comprised of 88 female patients aged between 30 and 80 with one sided secondary arm lymphedema after dissection of the axillary lymph nodes (level I or II according to the St. Gallen consensus conference) due to mammary cancer, who have been treated with combined decongestive therapy. All patients received the standard treatment - a combined decongestive therapy, comprising the manual lymphatic drainage on affected sites with consecutive bandaging of the affected arm and specially designed exercises and skin care from day 1 to day 20. The test group of patients received additionally Wobenzym at a dose of 5 coated tablets three times daily over 6.5 weeks.
Both treatment groups were well comparable. The median time between the lymph node dissection and the baseline visit was 47.5 months in the Wobenzym group and 48 months in the placebo group.
For measuring the indicator volume reduction in arm lymphedema, a Volometer was used. For the indicator tension, a four point rating scale was used. All the measurements were carried out on days 1, 9, 19, and 45 (final visit). CRP value was measured on days 1 and 19.
On the ill arm both groups showed a decrease of volume until visit 4 (day 45).
Both groups showed an almost identical course of the volumetric development between baseline visit and final end point visit, although there was a slight superiority of Wobenzym for the development between visit 3 and 4 with regard to the percent changes of –5% and more without statistical significance.
Both groups showed the greatest decrease of skinfold thickness between visits 2 and 3 with a very similar development between baseline visit and final visit with regard to the results of the ill arm.  The results of the percent changes from baseline with regard to visit 2, 3, and 4 showed a mean reduction by –29.84% for the verum group and –15.73% for placebo group. The development between visit 3 and 4 showed only slight superiority for the verum group. 
Both groups showed an almost similar decrease of tension in the ill arm until visit 4. A percent change of 100 % (total improvement) was reached by 62.79 % patients in the Wobenzym group and by 47.62 % patients in the placebo group. The percent changes between visits 3 and 4 – time where no concomitant combined decongestive therapy was applied – showed a clear superiority of the Wobenzym treated patients.
CRP was measured at visit 1 (baseline) and 3 (day 19). Wobenzym group showed better results than placebo group with regard to the CRP development between baseline visit and visit 3: out of 15 patients with high baseline findings five patients normalized in the Wobenzym group.
Out of 13 patients with high baseline findings in the placebo group, only one patient normalized. For patients with high CRP-values at baseline there has been mean percent change from baseline of –39.8%, while in the placebo group –17.4%.
There was a clear superiority for the verum group with regard to the CRP development between visit 1 and 3.
Overall, 15 adverse events were recorded, 7 for the verum group and 8 for the placebo group. The adverse events in the verum group were all gastrointestinal complaints of moderate intensity and rated as possibly (6 cases) or definitely (1 case) related to the study medication. All the patients showing adverse events completely recovered without sequelae.
All in all, the study failed to demonstrate efficacy in edema-related criteria (most likely due to extensive concomitant physical therapy in all patients) but demonstrated efficacy of Wobenzym with regard to the inflammation-related criteria. The inflammation-related criteria showed more than small superiority of Wobenzym. Moreover, for the subgroup “no chemotherapy” the inflammation-related criteria showed more than medium-sized superiority of Wobenzym. Reduced inflammatory tissue conditions are the basis for minimizing fibrosis thus preventing further inflammation and infection.
Poster Reference Number 45.